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British Journal of Radiology (2006) 79, e140-e144
© 2006 British Institute of Radiology
doi: 10.1259/bjr/44174031
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Spontaneous thrombosis of congenital cerebral arteriovenous malformation complicated by subdural collection: in utero detection with disappearance in infancy

B DeCesare, MD 2 M F Omojola, MB, FRCPC 1 E F Fogarty, MD 1 J C Brown, MD 1 and C Taylon, MD 3

Department of 1Radiology 2Creighton University Medical School3Department of Surgery, Creighton University Medical Center, Omaha, Nebraska, USA


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  		Figure 1. (a) Coronal cranial ultrasound at birth through the temporal lobes showing a 4 cm hyperechoic left temporal lobe mass. (b) Sagittal Doppler cranial ultrasound at birth through the left temporal lobe showing a left temporal lobe vascular mass. (c) Axial T1 weighted MR image at birth through the temporal lobes showing a well defined, ovoid, left temporal lobe mass of heterogeneous high signal, causing mild compression of the brain stem. (d) Post-intravenous gadolinium DTPA axial T1 weighted image through the lesion demonstrating intense enhancement and venous drainage into dilated transverse sinuses. (e) Coronal T2 weighted MR image at birth through the temporal lobes shows a round left temporal lobe mass of heterogeneous intensity with multiple tubular signal voids and large basilar and left posterior cerebral arteries. (f) Axial T2 weighted MR image at birth through the suprasellar cistern showing multiple tubular signal voids in the left temporal lobe. The left middle cerebral and left posterior cerebral arteries are large. (g) Axial T2 weighted MR image through the temporal lobes at the age of 4 months showing disappearance of the left temporal lobe arteriovenous malformation (AVM). No evidence of significant residual lesion. There is left temporal lobe volume loss. (h) Axial fluid attenuated inversion recovery (FLAIR) MR image through the level of the lateral ventricles at 4 months showing a small left frontal subdural collection. Left hemisphere atrophy is present. (i) Axial T2 weighted MR image through the suprasellar cistern at 7 months showing no evidence of a vascular malformation. There is left temporal lobe atrophy.

 




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