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Spontaneous regression of supratentorial intracerebral Langerhans' cell histiocytosis

¹ Department of Neuroimaging, King's College Hospital, Denmark Hill, London and ² Departments of Neuroradiology and Neurology, Atkinson Morley Hospital, Copse Hill, Wimbledon, London, UK

View larger version (103K): [in a new window]   Figure 1. Coronal T1 spin-echo (repetition time/echo time 550/20) post gadolinium-DTPA shows an homogeneously enhancing lesion within the left anteromedial temporal lobe with surrounding low T1 signal and sulcal effacement. A smaller enhancing lesion is seen in the right medial temporal lobe and there is evidence of meningeal enhancement.

View larger version (99K): [in a new window]   Figure 2. (a) 2 year later coronal fluid attenuated inversion recovery (repetition time/echo time/inversion time 7000/140/1950) shows a spherical, intermediate to low, heterogeneous signal lesion within the left temporal lobe with surrounding hyperintensity extending into the adjacent white matter. (b) Coronal T1 spin echo shows a lesion corresponding to an area of hyperdensity seen on the CT with a peripheral ring of hyperintensity and a central area of intermediate signal likely to represent intracellular methaemoglobin and central deoxygenated haemoglobin. A separate region of T1 hypointensity is again noted within the left temporal lobe.