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Endoanal ultrasound

G T Rottenberg, FRCR 1 and A B Williams, FRCS 2

Departments of 1 Radiology and 2 Surgery, Guy's and St Thomas' NHS Trust, Lambeth Palace Road, London SE1, UK



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Figure 1. Normal appearance of the male anal canal at the level of the puborectalis muscle. a, puborectalis; b, circular muscle of the rectum; c, prostate.

 


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Figure 2. Normal sphincter appearance in an adult male (mid anal canal). a, internal anal sphincter; b, longitudinal muscle; c, external anal sphincter.

 


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Figure 3. Normal sphincter appearance in an adult male (distal to the termination of the internal anal sphincter) demonstrating the normal subcutaneous portion of the external anal sphincter (arrow).

 


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Figure 4. Normal sphincter appearance in an adult female (mid anal canal). a, internal anal sphincter; b, longitudinal muscle; c, external anal sphincter. Note the poorer delineation between the external anal sphincter and the longitudinal layer than in the male patient in Figure 3Go.

 


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Figure 5. Prominent internal haemorrhoids. There is a hypoechoic focus in the subepithelial region (arrow) that should not be mistaken for a fistulous track.

 


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Figure 6. Marked thickening of the internal anal sphincter to 4.2 mm (arrow) in a 43-year-old patient with solitary rectal ulcer syndrome.

 


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Figure 7. Internal anal sphincter (IAS) atrophy in a 55-year-old man with passive incontinence. The IAS is thin but intact (arrow). The appearances are consistent with primary sphincter atrophy in the presence of normal pudendal nerve terminal motor latencies.

 


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Figure 8. Internal anal sphincter (IAS) disruption post anal stretch. The IAS is totally disrupted. The external anal sphincter is intact. Anal stretch procedures can produce single or multiple defects in the IAS.

 


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Figure 9. Internal anal sphincter (IAS) defect post surgery in a 27-year-old man with passive incontinence. There is a defect in the IAS between 6 O'clock and 11 O'clock (arrows) following surgery for haemorrhoids. The external anal sphincter is intact (arrowheads).

 


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Figure 10. Internal anal sphincter (IAS) defect post internal sphincterotomy. There is an IAS defect between 4 O'clock and 8 O'clock (arrow). The external anal sphincter appears attenuated but intact (arrowheads).

 


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Figure 11. Obstetric injury in a 32-year-old woman presenting with faecal urgency 6 months after a forceps delivery. There is a defect in the external sphincter between 1 O'clock and 3 O'clock (arrow). The normal external anal sphincter is shown (arrowhead).

 


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Figure 12. Severe obstetric trauma. There is a defect in the external anal sphincter between 9–12 O'clock and 2–3 O'clock (arrowheads). There is disruption of the internal anal sphincter (arrows).

 


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Figure 13. Obstetric injury and fistula formation. There is a defect in the anterior portion of the external anal sphincter with an anterior anal fistula (arrow). The tiny focus of increased reflectivity is from gas within pus in the fistula track.

 


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Figure 14. Obstetric injury, post repair. A defect in the internal anal sphincter remains between 10 O'clock and 3 O'clock. The external anal sphincter (EAS) has been repaired but there are still persistent changes in the EAS between 12 O'clock and 3 O'clock.

 


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Figure 15. Crohn's disease with recurrent fistula formation. There is disruption in the internal anal sphincter (arrow), which may be the result of previous sepsis compounded by surgery. There is evidence of an extra-sphincteric collection (arrowhead).

 


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Figure 16. Low intersphincteric collection between the internal and external anal sphincters (arrow).

 


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Figure 17. Low intersphincteric fistula in the (a) mid anal canal and (b) low anal canal. There is an intersphincteric collection low in the anal canal. The site of the internal opening is inferred by the point at which the collection enters the internal anal sphincter (a, arrow). The external anal sphincter appears intact.

 





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